Half life of prop is too long. It wouldn't be a bridge into another cycle but a bridge into a proper PCT. My hypothetical scenario was to run low dose of Var (10mg and tapering down) during PCT. However, you extend PCT 2 more weeks or for however long you run the low dose of var. So if you run nolvadex for 4 weeks you would instead run it 6 weeks with he first 2 weeks having a low dose of var that tapers down.
When you continue the same workout plan and diet you were running on cycle you are going to most likely become fat from the excessive caloric intake. And you would become overtrained from the volume and intensity of the same workout routine. This would result in you becoming fat and weak. I lower volume and try to maintain the weight lifted on cycle during PCT and I do maintain the caloric excess even though I start packing on fat.
The whole point of the low dose, low potency oral is to help prevent the rollback effect while slowly recovering. This happens because at low dosages certain orals will allow you to start recovering slowly. This would help you bridge into a proper PCT, possibly avoiding a severe loss of gains.
This is all hypothetical since I have not tried it yet. But I didn't come up with the idea on my own either.
I would be interested on hearing from somebody who has done this and had blood work done, so they can share their experience.
I think people are under the impression that I am suggesting a bridge by running anavar throughout entire PCT. I am suggesting running a low dose oral for 2 weeks post cycle and running a serm for 6 weeks post cycle. You would still be running a proper PCT unless you think 4 weeks is not long enough. I was suggesting adding however many weeks you run low dose oral to your amount of time using a serm. So if you normally run pct for say 6 weeks, and you take a low dose oral 2 weeks post cycle, you add those 2 weeks on to the 6 weeks you are running pct, which would be 8 weeks in all.
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Oh I understand what your referring to BUT there is one HUGE PROBLEM that most mates and physicians alike don't understand, YOU CAN'T TITRATE HTPA RECOVERY!!
It either secrets LH effectively (at appropriate doses, stimuli, timing and duration) or it does NOT! So when you add an exogenous AAS to the works during the HTPA restoration process (AKA PCT) your creating a physiologic dichotomy!
That is (AND THIS IS THE KEY TO UNDERSTAND WHAT'S HAPPENING ON A METABOLIC BASIS) YOUR USING ONE SUBSTANCE, LIKE VAR, WHICH DECREASES LH SECRETION AND THE OTHER, LIKE CLOMID, ENHANCES LH SECRETION. GOT IT?
MATE YOU SIMPLY CAN'T HAVE IT BOTH WAYS, BECAUSE THEY ARE MUTUALLY EXCLUSIVE!
However what can be done is use AAS with shorter half lives as bridge TO PCT. That will decrease the period in which there are NO AAS on board.
What types of AAS are optimally suited for PCT bridging? That will depend upon the potency, number AND DURATION of those cycles which proceeded it.
Does it work, you bet it does and in fact IME, for those heavy AAS users it's the ONLY way for them to tolerate the inevitable crash.
Look I'm talking about BB who have been "high end" cycling for years OR "bridging" between cycles for a longer period, never allowing complete HTPA recovery. (Quite a few need a several year "bridge" and begin TRT at that juncture,
with attempts at reversing the HTPA suppression a few months later)
The bottom line these mates have been on AAS for years and the only way many can tolerate the upcoming crash is a period of low dose AAS.
BEST
JIM