DHB (misnomer, it is 1-Testosterone) versus Tren: is it more like Primo or a "Tren lite"? [Author: Type-IIx]

talking shit behind a screen with a shitty physique.
Pot, meet kettle.

youre a nobody spouting nonsense, slamming your autistic mits on the keyboard because mommy isnt here to tell you how special and smart her baby boy is.

ballsy move calling out my physique looking like that.
 
You you have 0 comprehension skills. we are done here, I don’t debate stupidity
Thank you for plainly illustrating that you have nothing intelligent to contribute. Saves me a gram or two of glucose reading your posts. Ignored. For the record, God help anyone receiving treatment from you if you actually are a medical provider.
 
I've heard dumber shit today than PIP being associated with increased risk of infection/abscess. Nolij is actually totally correct in principle that there is more opportunity for abscess when, rather than being absorbed, a crystal forms in the tissue when the solute dissociates from the solution.

Of course, infection/abscess requires a bacterium.

In the case of a well-dissolved oil vehicle (that sometimes 1-Test is not because of its tendency to come out of solution), antimicrobial properties reduce this tendency.

Think about aqueous testosterone suspension, that is associated with PIP & abscesses as well. The key difference between aqueous testosterone suspension and its dissolution in tissues, formation of crystal aggregates, and risk of abscess is the absence of oil vehicle (and therefore antimicrobial properties).

Even though the risk remains very low of an abscess occurring with each injection of aqueous testosterone suspension as well; and the risk is virtually nil with well-dissolved 1-Test (i.e., using guaiacol as a solvent); the risk is intermediate for poorly dissolved 1-Test that doesn't hold in oil vehicle (i.e., test suspension > 1-Test without guaiacol or that otherwise "crashes" readily > everything else).

I am sure that the risk has never been detected with statistical significance because nobody has (nor will) investigate the phenomenon of abscesses caused by black market AAS to the extent that they'll have enough data to quantify abscess risk by compound/drug.
 
Last edited:
Is there any actual proof that DHB raises CRP or just anecdotes? In my own experience (N=1) low dose DHB did not increase CRP or ESR at all over 8 weeks. Also no PIP or any noticeable bad effects.
The only thing it increased in my bloodwork is liver enzymes.
Overall had a good experience and will definitely run it again.
 
Is there any actual proof that DHB raises CRP or just anecdotes? In my own experience (N=1) low dose DHB did not increase CRP or ESR at all over 8 weeks. Also no PIP or any noticeable bad effects.
The only thing it increased in my bloodwork is liver enzymes.
Overall had a good experience and will definitely run it again.
as with everything, id imagine it has to do with personal response. DHB can be prepared without guaiacol mostly pip free, and even if GC was used it seems like not everyone gets such a marked increase in inflammatory markers, although it seems to be relatively common.

so i think personal response is going to be dose specific, preparation specific, and personal tolerance specific
 
Is there any actual proof that DHB raises CRP or just anecdotes? In my own experience (N=1) low dose DHB did not increase CRP or ESR at all over 8 weeks. Also no PIP or any noticeable bad effects.
The only thing it increased in my bloodwork is liver enzymes.
Overall had a good experience and will definitely run it again.
Purely anecdotal! That's good to read, thank you for reporting this. ALT, AST, and GGT elevated (abnormally high? as in outside the reference range?)
 
Purely anecdotal! That's good to read, thank you for reporting this. ALT, AST, and GGT elevated (abnormally high? as in outside the reference range?)
ALT/AST were 90/121 in a reference range of 0-50, which shows some acute liver stress. GGT, which I understand is a marker of more permanent damage, was 40 in a reference range of 10-71, not elevated at all compared to previous bloodwork.
I consistenly run slighly elevated ALT/AST, also hard exercise elevates them, so not too worried.
CRB was 2.1 in a range of 0-5.
My cycle was TestC/NPP/DHB at 250/100/100.
 
But is it legit lol I literally got mine in the mail today. Missing the NPP I ordered but I am looking forward to trying it.

What was your experience w QSC dhb??
So far feels like I haven’t injected anything no pip no flu nothing at all I’m hungry hot but on ment too can’t say it it’s helping much though
 
So far feels like I haven’t injected anything no pip no flu nothing at all I’m hungry hot but on ment too can’t say it it’s helping much though

Still no pip? Ordered some the other day just waiting for it to come in.

Any feedback on performance would also be appreciated!
 
I got an abscess once in my life, it was from DHB, even though it was completely painless beforehand

overall, dhb gave me more side effects than all the steroids combined
 
Care to elaborate? What sides.
My LDL increased from 145 to 215, I stopped DHB and used EQ and ldl dropped to 150 in one month (i dont use statins)

It also gave me 140 BP when on other drugs my BP is about 125-130

In my opinion, we know far too little about how dhb works to use it
 
1-Testo produces as urine metabolites:

* 5α-dihydrotestosterone ("DHT")
So, this is super interesting.

You say it's the "primary metabolite", do we know if this conversion is large enough to create noticeable effects (dose dependent, of course).

The reason I ask is that having experimented extensively with DHB and followed results with both myself, clients, elsewhere, DHB (compared to say, Primo) seems to have effects that intuitively seem decidedly androgenic in nature. I can give specifics if curious.

Structurally, and what I've studied of DHB directly, it's been hard to rationalize chemically. I now wonder if many of the unique effects DHB seems to produce are really a product of DHT conversion. It would make a *lot* of sense, IMO.
 
So, this is super interesting.

You say it's the "primary metabolite", do we know if this conversion is large enough to create noticeable effects (dose dependent, of course).

The reason I ask is that having experimented extensively with DHB and followed results with both myself, clients, elsewhere, DHB (compared to say, Primo) seems to have effects that intuitively seem decidedly androgenic in nature. I can give specifics if curious.

Structurally, and what I've studied of DHB directly, it's been hard to rationalize chemically. I now wonder if many of the unique effects DHB seems to produce are really a product of DHT conversion. It would make a *lot* of sense, IMO.
IIn the only study on DHB metabolism, urinary DHT levels increased up to 7x above the norm in doping tests
 
So, this is super interesting.

You say it's the "primary metabolite", do we know if this conversion is large enough to create noticeable effects (dose dependent, of course).

The reason I ask is that having experimented extensively with DHB and followed results with both myself, clients, elsewhere, DHB (compared to say, Primo) seems to have effects that intuitively seem decidedly androgenic in nature. I can give specifics if curious.

Structurally, and what I've studied of DHB directly, it's been hard to rationalize chemically. I now wonder if many of the unique effects DHB seems to produce are really a product of DHT conversion. It would make a *lot* of sense, IMO.
It is. I don't know that it is dose dependent.

I have outlined my hypothesis about how it produces DHT here: [link]
 
It is. I don't know that it is dose dependent.

I have outlined my hypothesis about how it produces DHT here: [link]
I was wondering today, if DHB does significantly increase DHT levels, is it possible that it is extremely harmful to the prostate?

I saw a study that people given DHT had a significantly higher PSA than normal, although it was probably a study on older people.
 
I was wondering today, if DHB does significantly increase DHT levels, is it possible that it is extremely harmful to the prostate?

I saw a study that people given DHT had a significantly higher PSA than normal, although it was probably a study on older people.
It’s already 5 alpha reduced it won’t go to dht but dhb is more androgenic I believe than dht so it would be a little worse on the prostate…my blood pressure gets high on it but I don’t get many side effects I ran it several times blood was always ok but my last run i frontload a lot I did 1g per week first two weeks then down to about 600mg my blood pressure was high and my estrogen did get significantly lower but I think my test was at 375mg but that is almost a primobolan like effect on estrogen so I don’t know I wish I did more test and checked blood again
 
It’s already 5 alpha reduced it won’t go to dht but dhb is more androgenic I believe than dht so it would be a little worse on the prostate…w of the previous posts?
why don't you take the trouble to read at least a few of the previous posts?

If you would like to do interesting tests, I recommend checking the levels of DHT and E1 on DHB and inflammation markers
 

Attachments

  • IMG_20231110_195148 (1).png
    IMG_20231110_195148 (1).png
    211.8 KB · Views: 8
Back
Top