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Sir, if you don't mind I will write for you how I see cycle with gear which you have.
1-8wk
1)Testo P 100mg EOD
2)NNP 100mg EOD
3)Proviron 50 ED
1-9WK
1)anavar 30mg ED
HGH 5IU ED 5/2 1-9wk
HCG 500IU E4D 4-8wk
Ex 12.5 EOD
Caber 1 tab E14D
PCT
9-13 wk
1wk - Clomid 100mg + farestos 40mg
2wk - Clomid 100mg + farestos 40mg
3wk blood test (FSH/LH/testo total/e2/prolactin)
3wk - clomid 50mg + farestos 20mg
4wk clomid 50mg + farestos 20mg
But this is not best one cut cycle, just based on what you got.
Hello Sir, I will check!Hey pcom helped , I’m still waiting on my order... I ordered the 22 and it still hasn’t reached the states.
*bump the Test P up to 150 EOD --- for what?@Sargon
You should bump the Test P up to 150 EOD and run the whole cycle for 8-12 weeks. Possibly bump the anavar up to 50mg ED...but I wouldn't run the anavar longer than 6 weeks....preferbly at the back end (weeks 6-12, 4-10, or 2-8 depending on how long you run the cycle) don't know why he has it listed for 9 weeks.
Also discontinue the NPP 2 weeks before the Test P and you can start PCT 5 days to a week after your last pin.
Also I would recommend Nolvadex vs Fareston, people worry about the slight rise in progesterone but it is very very minimal and with Nolvadex is an antagonist as well.
It is more than sufficient for PCT and gyno related sides. It also is great at lowering cholesterol while Fareston is not.
*bump the Test P up to 150 EOD --- for what?
What the point to raise testo dosages when you have access to other AAS, without side effects like aromatisation or androgenic side effects like acne or alopecia? If you are not a pro atlet, you don't need big testo dosages, 200-400mg of short ester or 400-600mg of long esters more than enough. Large part of your cycle must be created by other AAS.
*and run the whole cycle for 8-12 weeks. --- Use short esters (often injections) for 12 weeks? I don't see reason for this. max 8 week or use long esters. Of course if you are not a fan of some kind of masochism.
*Possibly bump the anavar up to 50mg ED...but I wouldn't run the anavar longer than 6 weeks....preferbly at the back end (weeks 6-12, 4-10, or 2-8 depending on how long you run the cycle) don't know why he has it listed for 9 weeks. --- I also don't see reason to use protocols like a few weeks on the beginning or to the end, specially to anavar, better take anavar with small doses but during whole cycle rather than small parts of cycle with bid doses. Besides 20-30mg of anavar are almost never raise transaminases in most cases. We have atlets who use anavar more than 6 months without problems - transaminases in normal refs, ultrasound doesn't show toxic hepatitis or steatosis (fatty liver) - so in conclusion for liver better small doses of oral AAS longer time, than big doses but short time.
*don't know why he has it listed for 9 weeks. --- not so big necessary to use anavar untill 9 weeks, he can stop on week 8. No problem
*Also discontinue the NPP 2 weeks before the Test P and you can start PCT 5 days to a week after your last pin. --- for what? He can stop use both of them and start PCT week after last injections without any problems and hindrances.
*Also I would recommend nolvadex vs Fareston, people worry about the slight rise in progesterone but it is very very minimal and with Nolvadex is an antagonist as well. --- I recommend him to use Fareston not because he use nandrolones, reason was that fareston don't have so much side effects as nolva have. To our needs fareston more preferable as tamox, and if person can afford to buy fareston I recommend him to buy fareston.
*It is more than sufficient for PCT and gyno related sides. It also is great at lowering cholesterol while Fareston is not. It is more than sufficient for PCT and gyno related sides.
---Based from our practice/research fareston shows better result in gyno treatment and PCT than tamox and without side effects which occur when using tamox.
About lowering cholesterole you can read japan studies -"effects of toremifene (TOR) and tamoxifen (TAM) on serum lipids in postmenopasal patients with breast cancer" Briefly - fareston better than tamox.
By the way using SERMs like tamox or fareston to improve lipid panel when you are on cycle or PCT, not best idea, if you want normalize your lipids on cycle, you need statins (atorvastatin or rosuvastatin) if you are on PCT or just want preventive measures - you can use lovastatin.
Summarize:
Fareston chemical structure and pharmacology is close to tamox, but fareston has an chlorine atom, which is stabilizing molecule and decreasing the formation of aggressive metabolites that cause DNA damage.
Toxicity
Mutagenicity in vivo
Tamox - Causes chromosome damage in liver cells.
Fareston - NO.
Education DNA adducts
Tamox - Induces a significant amount adducts of DNA in liver cells (transversions, inversions and deletion) - genotoxic. Formation of adducts in leukocytes and endometrium.
Fareston - NO.
Vision
Tamox - cataracts, less other - retinopathy.
Fareston - hardly ever.
Carcinogenicity
Tamox - induced hepatic cell carcinoma, endometrial cancer, GI tract cancer.
Fareston - NO.
https://www.suppreviewers.com/torem-vs-nolva-pct/
Toremifene Improves Lipid Profiles in Men Receiving Androgen-Deprivation Therapy for Prostate Cancer: Interim Analysis of a Multicenter Phase III Study
Matthew R. Smith, S. Bruce Malkowicz, Franklin Chu, John Forrest, Paul Sieber, K. Gary Barnette, Domingo Rodriquez, and Mitchell S. Steiner
In the subset of nonusers of cholesterol-lowering medications, total cholesterol decreased by 0.2% ± 2.2% from baseline to month 12 in the placebo group and decreased by 9.2% ± 1.7% in the toremifene group (P = .002). LDL cholesterol increased by 3.1% ± 3.3% in the placebo group and decreased by 11.6% ± 3.1% in the toremifene group (P = .001). HDL cholesterol decreased by 6.7% ± 1.9% in the placebo group and increased by 3.2% ± 3.2% in the toremifene group (P= .005). Triglycerides increased by 12.4% ± 6.9% in the placebo group and decreased by 14.6% ± 5.2% in the toremifene group (P = .045). The ratio of total to HDL cholesterol increased by 8.9% ± 3.1% in the placebo group and decreased by 9.7% ± 2.3% in the toremifene group (P < .001).
I understand you are trying to help but you are giving out wrong information.
mands
Format: Abstract
Send to
Clin Chim Acta. 1993 Dec 31;223(1-2):43-52.
Effect of tamoxifen on serum cholesterol and lipoproteins during chemohormonal therapy.
Dnistrian AM1, Schwartz MK, Greenberg EJ, Smith CA, Schwartz DC.
Author information
Abstract
The effect of tamoxifen on serum cholesterol, high density lipoprotein cholesterol (HDL-cholesterol), low density lipoprotein cholesterol (LDL-cholesterol) and the ratio of LDL-cholesterol to HDL-cholesterol (LDL-C/HDL-C) was investigated in breast cancer patients undergoing therapy for advanced disease. Longitudinal studies in 24 patients treated with tamoxifen (10 mg, twice daily) indicated average decreases in total serum cholesterol (17%) and LDL-cholesterol (27%), whereas the effect of tamoxifen on HDL-cholesterol varied with the individual patient. There was a significant decrease in the LDL-C/HDL-C ratio (33%) consistent with a decreased risk for coronary artery disease. This beneficial influence of tamoxifen on risk factors associated with cardiovascular disease was evident in both premenopausal and postmenopausal patients whether tamoxifen was administered alone or in combination with cytotoxic chemotherapy.
mands
Pich. Are you actually going to debate with a rep that has probably trained and has taken AAS longer then both our ages combined ? Haha.
Nothing wrong with healthy debate + discussion that's how we all learn -- myself included.
Go to the thread I quoted @mands on and check it out
I feel the reason he doesn’t need 2 weeks longer for pct is because npp has a short active life...
Now if he were taking deca being the long ester. I would definitely recommend two weeks prior to pct.
I have read that far is much less potent as far as sides then nolva and tami
Would a week be good though ? It’s active life is like 5 days
Pich. Are you actually going to debate with a rep that has probably trained and has taken AAS longer then both our ages combined ? Haha.
Reps are mostly pretty knowledgeable in their fields and I tend to trust the ones pushing less product than more and especially not pushing a brand. The only thing I disagree with @Pharmacom Helper on is that everyone’s different and their body’s all process stuff based on health, diet, metabolism, and lots of other factors like existing high BP ext.. So giving a blind dose recommendation can be tough and mostly just used as a guideline anyhow. It’s a good guideline but I’ve never found any dose of Anavar less than 40mg to 50mg ED to be useful. That’s just me though. Like I said, everyone’s different.
just because he is a rep doesn't mean anything. reps are sales people like used car salesman they may talk the talk but cant even change a tire.
*bump the Test P up to 150 EOD --- for what?
What the point to raise testo dosages when you have access to other AAS, without side effects like aromatisation or androgenic side effects like acne or alopecia? If you are not a pro atlet, you don't need big testo dosages, 200-400mg of short ester or 400-600mg of long esters more than enough. Large part of your cycle must be created by other AAS.
*and run the whole cycle for 8-12 weeks. --- Use short esters (often injections) for 12 weeks? I don't see reason for this. max 8 week or use long esters. Of course if you are not a fan of some kind of masochism.
*Possibly bump the anavar up to 50mg ED...but I wouldn't run the anavar longer than 6 weeks....preferbly at the back end (weeks 6-12, 4-10, or 2-8 depending on how long you run the cycle) don't know why he has it listed for 9 weeks. --- I also don't see reason to use protocols like a few weeks on the beginning or to the end, specially to anavar, better take anavar with small doses but during whole cycle rather than small parts of cycle with bid doses. Besides 20-30mg of anavar are almost never raise transaminases in most cases. We have atlets who use anavar more than 6 months without problems - transaminases in normal refs, ultrasound doesn't show toxic hepatitis or steatosis (fatty liver) - so in conclusion for liver better small doses of oral AAS longer time, than big doses but short time.
*don't know why he has it listed for 9 weeks. --- not so big necessary to use anavar untill 9 weeks, he can stop on week 8. No problem
*Also discontinue the NPP 2 weeks before the Test P and you can start PCT 5 days to a week after your last pin. --- for what? He can stop use both of them and start PCT week after last injections without any problems and hindrances.
*Also I would recommend nolvadex vs Fareston, people worry about the slight rise in progesterone but it is very very minimal and with Nolvadex is an antagonist as well. --- I recommend him to use Fareston not because he use nandrolones, reason was that fareston don't have so much side effects as nolva have. To our needs fareston more preferable as tamox, and if person can afford to buy fareston I recommend him to buy fareston.
*It is more than sufficient for PCT and gyno related sides. It also is great at lowering cholesterol while Fareston is not. It is more than sufficient for PCT and gyno related sides.
---Based from our practice/research fareston shows better result in gyno treatment and PCT than tamox and without side effects which occur when using tamox.
About lowering cholesterole you can read japan studies -"effects of toremifene (TOR) and tamoxifen (TAM) on serum lipids in postmenopasal patients with breast cancer" Briefly - fareston better than tamox.
By the way using SERMs like tamox or fareston to improve lipid panel when you are on cycle or PCT, not best idea, if you want normalize your lipids on cycle, you need statins (atorvastatin or rosuvastatin) if you are on PCT or just want preventive measures - you can use lovastatin.
Summarize:
Fareston chemical structure and pharmacology is close to tamox, but fareston has an chlorine atom, which is stabilizing molecule and decreasing the formation of aggressive metabolites that cause DNA damage.
Toxicity
Mutagenicity in vivo
Tamox - Causes chromosome damage in liver cells.
Fareston - NO.
Education DNA adducts
Tamox - Induces a significant amount adducts of DNA in liver cells (transversions, inversions and deletion) - genotoxic. Formation of adducts in leukocytes and endometrium.
Fareston - NO.
Vision
Tamox - cataracts, less other - retinopathy.
Fareston - hardly ever.
Carcinogenicity
Tamox - induced hepatic cell carcinoma, endometrial cancer, GI tract cancer.
Fareston - NO.
@ironwill1951 that is good advice and people should listen to it because I know we have all run across fools that are reps many times.
After all, it is not that easy for AAS sources to find or afford people with degrees in chemistry/biochem, molecular biology, medicine etc AND years of experience -- AND that are interested to answer questions online (small sources simply would not have access or be able to afford such employees).
However, while that may sometimes be the case, @Pharmacom Helper is a wealth of knowledge and experience -- I can tell from multiple discussion that he has a solid formal education (maybe in the medical field? I am not sure of it) and a lot of hands on life experience (likely helps coach the Pharmacom Athletes team).
While, sure, like with any two people he and I may not always agree perfectly 100% of the time (for example, I suggest other supplements some times and also I avoid statins -- we each know some different things), I greatly respect that he has VERY solid information and I know for a fact we can all learn a lot from his experience.
@Pharmacom Helper is NOT a typical rep; he is a full-time employee, an awesome asset and wealth of great information.
Thank you for taking time time and sharing all this advice my friend -- I enjoy getting a chance to read something instead of type haha