Acne

I'm lucky enough to be married to a woman who has a natural cure for everything. She has helped me start growing my hair back, and boot the acne. Bentonite Clay works wonders for a lot of things. It eats the hell out of acne, and removes heavy metals from your body. Which is awesome, considering UGL's might have trace amounts of heavy metals in their raws. Most high end spas use it as a base for all their body wraps, and masks. It actually separates the good from the bad, and releases the good back into your system. It is some strong shit though, so I only use it once a week. I also use African black soap to wash with every day. You can get both on Amazon cheap.

http://www.amazon.com/100-Pure-Bentonite-Powder-Pounds/dp/B001EECRLQ

http://www.amazon.com/African-Black-Soap-100-Pure/dp/B005ELPNLG
 
Also, I do not work for, or represent either of those companies. I do not care if you order them, but I promise that they work. I'm the whitest person you would ever meet, and I'm uber sensitive to everything. That shit pulled so much stuff out of my skin, it was scary. I looked like a splotchy freak right after, but it went away after a few moments.
 
someone literally just told me he heard on the radio about rubbing garlic cloves on your acne for 1 minute then wait five minutes then wash it off with warm water and i guess its supposed to help with acne?!? o_O wtf....
 
someone literally just told me he heard on the radio about rubbing garlic cloves on your acne for 1 minute then wait five minutes then wash it off with warm water and i guess its supposed to help with acne?!? o_O wtf....

I don't know about the garlic, I've never tried it. It makes sense to me that it would work though. I Actually use the products I posted about. The black soap was too drying for my wife to use all the time, but my skin is oily'er than hers.
 
I don't know about the garlic, I've never tried it. It makes sense to me that it would work though. I Actually use the products I posted about. The black soap was too drying for my wife to use all the time, but my skin is oily'er than hers.
I don't believe in everything the "natural" folks out there preach. However, I also don't believe that something will not work without FDA approval. There is some good stuff out there that is in nature.
 
@A-a-Ron Hell, she tried to make me stop eating meat, and that's where I drew the line. She's definitely way more into it than I am, but that clay and soap worked wonders for me.
 
Zomorodian K, Rahimi MJ, Taheri M, Ghanbari Asad A, Khani S, et al. The cutaneous bacterial microflora of the bodybuilders using anabolic-androgenic steroids. Jundishapur J Microbiol. 2015;8(1):e12269. http://jjmicrobiol.com/?page=article&article_id=12269

BACKGROUND: Anabolic-androgenic steroids (AAS) abuse by the athletes has dramatically increased during the recent decades. These substances might increase the skin lipids and enhance the cutaneous microbial proliferation.

OBJECTIVES: The current study aimed to investigate the potential side effects of AAS on the bacterial microflora colonization of the bodybuilders` skin.

PATIENTS AND METHODS: The skin samples of 94 male bodybuilders (71 AAS users, 23 non-AAS users) and 46 subjects of the control group, with similar gender and age, were cultured and incubated in both aerobic condition to isolate Staphylococcus aureus and anaerobic condition for Propionibacterium acnes. The isolated bacteria were identified by standard microbiological techniques.

RESULTS: The skin lesions were more frequent in the body builders than the controls. Moreover, statistically significant differences were also observed in skin lesions among the AAS users and the non-AAS user athletes. The prevalence of S. aureus and P. acnes in the athletes was higher than that of the control group. In addition, there was a significant difference in distribution of P. acnes between the bodybuilders who used AAS and those who did not.

CONCLUSIONS: A higher number of bacterial flora was found in the bodybuilders particularly those using AAS in comparison to the controls, which might be due to the influence of these AAS on the skin microflora and transmission of the bacteria through the direct contact of the naked skin with the exercise instruments.
 
Zomorodian K, Rahimi MJ, Taheri M, Ghanbari Asad A, Khani S, et al. The cutaneous bacterial microflora of the bodybuilders using anabolic-androgenic steroids. Jundishapur J Microbiol. 2015;8(1):e12269. http://jjmicrobiol.com/?page=article&article_id=12269

BACKGROUND: Anabolic-androgenic steroids (AAS) abuse by the athletes has dramatically increased during the recent decades. These substances might increase the skin lipids and enhance the cutaneous microbial proliferation.

OBJECTIVES: The current study aimed to investigate the potential side effects of AAS on the bacterial microflora colonization of the bodybuilders` skin.

PATIENTS AND METHODS: The skin samples of 94 male bodybuilders (71 AAS users, 23 non-AAS users) and 46 subjects of the control group, with similar gender and age, were cultured and incubated in both aerobic condition to isolate Staphylococcus aureus and anaerobic condition for Propionibacterium acnes. The isolated bacteria were identified by standard microbiological techniques.

RESULTS: The skin lesions were more frequent in the body builders than the controls. Moreover, statistically significant differences were also observed in skin lesions among the AAS users and the non-AAS user athletes. The prevalence of S. aureus and P. acnes in the athletes was higher than that of the control group. In addition, there was a significant difference in distribution of P. acnes between the bodybuilders who used AAS and those who did not.

CONCLUSIONS: A higher number of bacterial flora was found in the bodybuilders particularly those using AAS in comparison to the controls, which might be due to the influence of these AAS on the skin microflora and transmission of the bacteria through the direct contact of the naked skin with the exercise instruments.
When they say "lesions," are they speaking of Nevi? or acne?
 
Blasiak RC, Stamey CR, Burkhart CN, Lugo-Somolinos A, Morrell DS. High-Dose Isotretinoin Treatment and the Rate of Retrial, Relapse, and Adverse Effects in Patients With Acne Vulgaris . JAMA Dermatol. 2013;149(12):1392-1398. http://archderm.jamanetwork.com/article.aspx?articleid=1764027

Importance Isotretinoin is the most effective treatment for acne. The ideal dosing regimen is unknown.

Objective To determine the rates of relapse of acne vulgaris and retrial of isotretinoin after high cumulative-dose treatment and the changes to the adverse effect profile.

Design, Setting, and Participants A prospective, observational, intervention study was conducted from August 1, 2008, to August 31, 2010, in a single academic tertiary care center with multiple providers. A total of 180 patients with acne resistant to other treatments were enrolled. Of these, 116 participated in the 12-month follow-up survey, for a response rate of 64.4%.

Exposure Patients received isotretinoin, with dosing based on the providers’ judgment. Patients were divided into 2 groups on the basis of cumulative dosing (<220 mg/kg and ≥220 mg/kg).

Main Outcomes and Measures Relapse (treatment with a prescription topical or oral acne medication after a course of isotretinoin) or retrial (retreatment with isotretinoin) at 12-month follow-up and adverse effects experienced during and after 12 months of treatment.

Results The mean age of the participants was 19.3 years, 51.9% were female, and 74.1% were white. At 12 months’ follow-up, 97.4% of the patients reported that their acne was improved. Overall, acne in 32.7% of patients in the study relapsed at 12 months, and 1.72% of the patients required a retrial. In the lower-dose treatment group (<220 mg/kg), the relapse rate was 47.4% (95% CI, 32.3%-63.0%) compared with 26.9% (95% CI, 18.3%-37.8%) in the high-dose group (P = .03). Almost 100% of the patients in both treatment groups developed cheilitis and xerosis during treatment. Retinoid dermatitis was significantly more common in the high-dose treatment group (53.8% vs 31.6%; P = .02). None of the other adverse effects was significantly different between the 2 groups.

Conclusions and Relevance The dosing regimen used in the present study is considerably higher than that used in previous studies of isotretinoin. At 1 year after completion of isotretinoin treatment, we found that patients receiving 220 mg/kg or more had a significantly decreased risk of relapse. Rash was the only adverse effect that was significantly more common in the high-dose group during treatment. This study suggests that significantly higher doses of isotretinoin are effective for treating acne and decreasing relapse rates without increasing adverse effects.
 
Blasiak RC, Stamey CR, Burkhart CN, Lugo-Somolinos A, Morrell DS. High-Dose Isotretinoin Treatment and the Rate of Retrial, Relapse, and Adverse Effects in Patients With Acne Vulgaris . JAMA Dermatol. 2013;149(12):1392-1398. http://archderm.jamanetwork.com/article.aspx?articleid=1764027

Importance Isotretinoin is the most effective treatment for acne. The ideal dosing regimen is unknown.

Objective To determine the rates of relapse of acne vulgaris and retrial of isotretinoin after high cumulative-dose treatment and the changes to the adverse effect profile.

Design, Setting, and Participants A prospective, observational, intervention study was conducted from August 1, 2008, to August 31, 2010, in a single academic tertiary care center with multiple providers. A total of 180 patients with acne resistant to other treatments were enrolled. Of these, 116 participated in the 12-month follow-up survey, for a response rate of 64.4%.

Exposure Patients received isotretinoin, with dosing based on the providers’ judgment. Patients were divided into 2 groups on the basis of cumulative dosing (<220 mg/kg and ≥220 mg/kg).

Main Outcomes and Measures Relapse (treatment with a prescription topical or oral acne medication after a course of isotretinoin) or retrial (retreatment with isotretinoin) at 12-month follow-up and adverse effects experienced during and after 12 months of treatment.

Results The mean age of the participants was 19.3 years, 51.9% were female, and 74.1% were white. At 12 months’ follow-up, 97.4% of the patients reported that their acne was improved. Overall, acne in 32.7% of patients in the study relapsed at 12 months, and 1.72% of the patients required a retrial. In the lower-dose treatment group (<220 mg/kg), the relapse rate was 47.4% (95% CI, 32.3%-63.0%) compared with 26.9% (95% CI, 18.3%-37.8%) in the high-dose group (P = .03). Almost 100% of the patients in both treatment groups developed cheilitis and xerosis during treatment. Retinoid dermatitis was significantly more common in the high-dose treatment group (53.8% vs 31.6%; P = .02). None of the other adverse effects was significantly different between the 2 groups.

Conclusions and Relevance The dosing regimen used in the present study is considerably higher than that used in previous studies of isotretinoin. At 1 year after completion of isotretinoin treatment, we found that patients receiving 220 mg/kg or more had a significantly decreased risk of relapse. Rash was the only adverse effect that was significantly more common in the high-dose group during treatment. This study suggests that significantly higher doses of isotretinoin are effective for treating acne and decreasing relapse rates without increasing adverse effects.
Dr. Scally,

Can steroid use increase Nevi (lesions, brown spots) on the skin?
 
I mix together in a spray bottle, rubbing alcohol, which hazel, and astringent and spray my back and acne prone spots down after I shower. That and tanning works well for me.
 
I mix together in a spray bottle, rubbing alcohol, which hazel, and astringent and spray my back and acne prone spots down after I shower. That and tanning works well for me.
Is that painful? Ive been getting all kinds if nasty acne lately... fpr 6wks on cycle I had nothing, then upper arms and shoulders broke out crazy. That kindof faded away and then forearms started breaking out but not as bad. Then all of a sudden my lower back on both sides was riddled with acne.. and now on wk11, I just started getting a bit of acne right above my ass crack.. I wonder why it hits in different time frames instead of all at once.
 
Is that painful? Ive been getting all kinds if nasty acne lately... fpr 6wks on cycle I had nothing, then upper arms and shoulders broke out crazy. That kindof faded away and then forearms started breaking out but not as bad. Then all of a sudden my lower back on both sides was riddled with acne.. and now on wk11, I just started getting a bit of acne right above my ass crack.. I wonder why it hits in different time frames instead of all at once.

No it's not painful at all. If I do it on a tanning day it stings for a min. Besides you stick pins in your ass lol can't be worried about a little alcohol sing.
 
Acne Fulminans Induced By Anabolic Steroids

10454


Clinical images of the case patient. A and B, The patient at the time of his first consultation shows severe acne fulminans with inflammatory nodules, pustules, and deep hemorrhagic ulcerations in the chest area and back. C and D, The same patient 6 months after treatment with oral prednisolone and isotretinoin shows extensive scarring.


A 22-year-old male amateur bodybuilder presented to our clinic with a 3-month history of severe acne lesions on his upper trunk and face, accompanied by arthralgia of several joints. He reported the use of anabolic androgenic steroids (AAS) (testosterone enanthate, trenbolone acetate, drostanolone propionate, and methandrostenolone) for 3 months to increase his muscle mass. Shortly after he discontinued AAS intake, he developed severe inflammatory acne with painful rupturing and draining inflammatory nodules, pustules, and hemorrhagic ulcerations on his upper trunk and face (Figure, A and B). Moreover, he described an immobilizing arthralgia of his right ankle and both shoulder joints, as well as general symptoms including fatigue and a 15-kg weight loss over the 6 weeks prior to presentation.

Treatment with several antibiotics had been attempted, including erythromycin, clindamycin, doxycycline, and flucloxacillin, but his condition did not improve. Treatment with isotretinoin, 20 mg/d, was stopped after 4 weeks because of an aggravation of skin lesions. He presented to our clinic while being treated with oral doxycycline, 100 mg/d, and prednisolone, 40 mg/d, for 1 week. Enlarged lymph nodes were palpable in the groin; fever was absent.

Laboratory examination showed leukocytosis (white blood cell count, 19 400/?L) and an elevated C-reactive protein concentration of 52.3 mg/L. (To convert white blood cells to number of cells × 109/L, multiply by 0.001; to convert C-reactive protein to nanomoles per liter, multiply by 9.524.) Magnetic resonance imaging of the patient's right ankle joint revealed soft-tissue edema and articular effusion. The diagnosis of AAS-induced acne fulminans was made, characterized by sudden onset, ulceration of lesions, fever, polyarthralgia, weight loss, and failure of usual antibiotic therapy.

In our clinic, doxycycline and prednisolone treatment were discontinued. The patient was treated locally with debridements, antiseptic wound dressings, and benzoyl peroxide ointments. A new systemic therapy with isotretinoin, 60 mg/d, was started 3 days later. Arthralgia was controlled by treatment with oral diclofenac, 100 mg/d. A continuous improvement with reepithelialization of ulcerations and reduction of purulent lesions and nodules was noted. After 3 months, the isotretinoin dose was reduced, after 6 months, isotretinoin therapy was discontinued. However, the formation of extensive and partly hypertrophic scars could not be prevented (Figure, C and D).

The illegal use of AAS is increasingly common even among leisure sports athletes. Doping prevalence among college athletes in the United States reportedly is 17% to 20%; among male amateur bodybuilders it is even as high as 80%. In addition to cardiovascular, hepatocellular, and psychological adverse effects, 43% of patients who abuse AAS develop androgen-induced acne. As derivatives of the hormone testosterone, AAS lead to hypertrophy of the sebaceous glands, increased sebum production, and increased density of the Propionibacterium acnes population.

Our patient developed AAS-induced acne fulminans with the typical unresponsiveness to systemic antibiotics. After initial therapy with oral prednisolone, 0.5 mg/kg, and debridements, a clinical response was achieved with isotretinoin, 0.75 mg/kg.

In conclusion, it is important for health care providers to keep in mind that androgen-induced acne is one of the most frequent symptoms of AAS abuse. The most important measure is the immediate termination of AAS administration. Increased public education is needed to curb AAS abuse and associated health risks.


Kraus SL, Emmert S, Schon MP, Haenssle HA. The dark side of beauty: acne fulminans induced by anabolic steroids in a male bodybuilder. Arch Dermatol 2012;148(10):1210-2. JAMA Network | Archives of Dermatology | The Dark Side of Beauty: Acne Fulminans Induced by Anabolic Steroids in a Male Bodybuilder
Correct me if I am wrong but isn't DHT the hormone responsible for hormonal acne? Maybe his DHT levels are messed up? It can happen with out the use of AAS right?

Or If the liver is not working right the body can sweat out toxins and that can clog the pores and cause acne. Right?
 
Acne Fulminans Induced By Anabolic Steroids

10454


Clinical images of the case patient. A and B, The patient at the time of his first consultation shows severe acne fulminans with inflammatory nodules, pustules, and deep hemorrhagic ulcerations in the chest area and back. C and D, The same patient 6 months after treatment with oral prednisolone and isotretinoin shows extensive scarring.


A 22-year-old male amateur bodybuilder presented to our clinic with a 3-month history of severe acne lesions on his upper trunk and face, accompanied by arthralgia of several joints. He reported the use of anabolic androgenic steroids (AAS) (testosterone enanthate, trenbolone acetate, drostanolone propionate, and methandrostenolone) for 3 months to increase his muscle mass. Shortly after he discontinued AAS intake, he developed severe inflammatory acne with painful rupturing and draining inflammatory nodules, pustules, and hemorrhagic ulcerations on his upper trunk and face (Figure, A and B). Moreover, he described an immobilizing arthralgia of his right ankle and both shoulder joints, as well as general symptoms including fatigue and a 15-kg weight loss over the 6 weeks prior to presentation.

Treatment with several antibiotics had been attempted, including erythromycin, clindamycin, doxycycline, and flucloxacillin, but his condition did not improve. Treatment with isotretinoin, 20 mg/d, was stopped after 4 weeks because of an aggravation of skin lesions. He presented to our clinic while being treated with oral doxycycline, 100 mg/d, and prednisolone, 40 mg/d, for 1 week. Enlarged lymph nodes were palpable in the groin; fever was absent.

Laboratory examination showed leukocytosis (white blood cell count, 19 400/?L) and an elevated C-reactive protein concentration of 52.3 mg/L. (To convert white blood cells to number of cells × 109/L, multiply by 0.001; to convert C-reactive protein to nanomoles per liter, multiply by 9.524.) Magnetic resonance imaging of the patient's right ankle joint revealed soft-tissue edema and articular effusion. The diagnosis of AAS-induced acne fulminans was made, characterized by sudden onset, ulceration of lesions, fever, polyarthralgia, weight loss, and failure of usual antibiotic therapy.

In our clinic, doxycycline and prednisolone treatment were discontinued. The patient was treated locally with debridements, antiseptic wound dressings, and benzoyl peroxide ointments. A new systemic therapy with isotretinoin, 60 mg/d, was started 3 days later. Arthralgia was controlled by treatment with oral diclofenac, 100 mg/d. A continuous improvement with reepithelialization of ulcerations and reduction of purulent lesions and nodules was noted. After 3 months, the isotretinoin dose was reduced, after 6 months, isotretinoin therapy was discontinued. However, the formation of extensive and partly hypertrophic scars could not be prevented (Figure, C and D).

The illegal use of AAS is increasingly common even among leisure sports athletes. Doping prevalence among college athletes in the United States reportedly is 17% to 20%; among male amateur bodybuilders it is even as high as 80%. In addition to cardiovascular, hepatocellular, and psychological adverse effects, 43% of patients who abuse AAS develop androgen-induced acne. As derivatives of the hormone testosterone, AAS lead to hypertrophy of the sebaceous glands, increased sebum production, and increased density of the Propionibacterium acnes population.

Our patient developed AAS-induced acne fulminans with the typical unresponsiveness to systemic antibiotics. After initial therapy with oral prednisolone, 0.5 mg/kg, and debridements, a clinical response was achieved with isotretinoin, 0.75 mg/kg.

In conclusion, it is important for health care providers to keep in mind that androgen-induced acne is one of the most frequent symptoms of AAS abuse. The most important measure is the immediate termination of AAS administration. Increased public education is needed to curb AAS abuse and associated health risks.


Kraus SL, Emmert S, Schon MP, Haenssle HA. The dark side of beauty: acne fulminans induced by anabolic steroids in a male bodybuilder. Arch Dermatol 2012;148(10):1210-2. JAMA Network | Archives of Dermatology | The Dark Side of Beauty: Acne Fulminans Induced by Anabolic Steroids in a Male Bodybuilder
 
Back
Top